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Herpes simplex virus-1 (HSV-1) infection of cultured human neural cells causes the accumulation of a host cell-encoded nuclear protein identified as a 57,000 mol.wt. stress protein by monoclonal antibody TI56. This protein is cell cycle-related in fibroblasts, may mediate host cell control during HSV infection and could play a role in the regulation of HSV latency. © 1985.

Original publication

DOI

10.1016/0304-3940(85)90484-7

Type

Journal article

Journal

Neuroscience Letters

Publication Date

11/11/1985

Volume

61

Pages

321 - 326