Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

Renal toxicity was assessed in 19 patients receiving methyl acetylenic putrescine (MAP), an irreversible inhibitor of ornithine decarboxylase. Patients received 250 mg t.d.s. for up to 13 weeks. This dose effectively inhibited the target enzyme, as shown by elevations in decarboxylated S-adenosyl methionine levels. No significant nephrotoxicity was observed in these patients as determined by plasma urea, creatinine and creatinine clearance measurements, although minor elevations of the urinary enzymes lactate dehydrogenase, N-acetyl-beta-glucosaminidase, alkaline phosphatase and alanine aminopeptidase were observed. As this could represent sub-clinical renal damage, caution should be exercised when using MAP in combination with other cytotoxic drugs.

Type

Journal article

Journal

Cancer Chemother Pharmacol

Publication Date

1990

Volume

26

Pages

65 - 66

Keywords

Acetylglucosaminidase, Adult, Aged, Alkaline Phosphatase, Alkynes, Aminopeptidases, CD13 Antigens, Creatinine, Diamines, Drug Evaluation, Humans, Kidney, L-Lactate Dehydrogenase, Microvilli, Middle Aged, Ornithine Decarboxylase, Ornithine Decarboxylase Inhibitors, Urea