Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

Base excision repair is the major pathway for the repair of oxidative DNA damage in human cells that is initiated by a damage-specific DNA glycosylase. In human cells, the major DNA glycosylases for the excision of oxidative base damage are OGG1 and NTH1 that excise 8-oxoguanine and oxidative pyrimidines, respectively. We find that both enzymes have limited activity on DNA lesions located in the vicinity of the 3' end of a DNA single-strand break, suggesting that other enzymes are involved in the processing of such lesions. In this study, we identify and characterize NEIL1 as a major DNA glycosylase that excises oxidative base damage located in close proximity to the 3' end of a DNA single-strand break.

Original publication

DOI

10.1093/nar/gki816

Type

Journal article

Journal

Nucleic Acids Res

Publication Date

2005

Volume

33

Pages

4849 - 4856

Keywords

Base Sequence, DNA Damage, DNA Glycosylases, DNA Repair, Deoxyribonuclease (Pyrimidine Dimer), Guanine, Humans, Oligonucleotides, Oxidative Stress, Substrate Specificity, Uracil